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Limitations of early transgenic models included a lack of an efficient method for gene deletion but rather for addition or misexpression and an absence of inducible systems. This absence of extensive banked tumor was one factor contributing to exclusion of SCLC from The Cancer Genome Atlas TCGA efforts, which thus far has comprehensively evaluated hundreds of squamous carcinomas and adenocarcinomas of the lung 22 , GEMMs: conditional mouse models of SCLC Genes can be conditionally deleted in mice utilizing specific methods, such as administering cell type-specific adenoviral vectors or by crossing to recombinant mice. Activity and safety of nivolumab, an anti-PD-1 immune checkpoint inhibitor, for patients with advanced, refractory squamous non-small-cell lung cancer CheckMate : a phase 2, single-arm trial. Two independent, comprehensive genomic studies, which included exome, whole genome, transcriptome, and copy number alteration data from primary SCLC patient samples together over samples have provided some initial insights into the fuller landscape of genetic alterations in this disease 26 , Histone deacetylase inhibitors in cancer therapy. Loss of p accelerates tumor development in a mouse model for human small-cell lung carcinoma. Twice-daily compared with once-daily thoracic radiotherapy in limited small-cell lung cancer treated concurrently with cisplatin and etoposide. Interestingly, effector T cells associated with cytolytic responses are significantly higher in the peripheral blood of patients with LS-SCLC compared to those with ES-SCLC and in long term disease-free survivors relative to those with recurrent disease

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